Guide To Lactic Acidosis Symptoms

Loss Of Appetite

Lactic acidosis patients may present with a loss of appetite due to their condition. Numerous factors contribute to an affected individual's appetite loss. Lactic acidosis causes the acidity of the blood to become too high, inducing vascular constriction. Blood vessel constriction can cause issues all around the body, but particularly in the digestive tract. The lack of blood supply due to constricted vessels causes food to move through the gastrointestinal tract slower than usual. When the food stagnates or moves slowly, the affected individual will not feel hungry from one meal to the next. Lactic acidosis can be the result of bacteria growth and colonization in the colon as a result of a short small intestine that cannot fully digest glucose. The bacteria in the large intestine overproduce due to the abundance of glucose and may invade into the small intestine. Bacterial infection of the small intestine causes inflammation of these tissues. This type of inflammation and toxicity in the small bowel often induces nausea and vomiting in affected individuals, effectively reducing their appetite. Pancreatitis, the inflammation of the pancreas, has been implicated as a cause for the development of lactic acidosis, which also produces nausea, vomiting, and appetite loss.

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Diarrhea

An individual affected by lactic acidosis may present with diarrhea as a symptom of their condition. Diarrhea is a symptom often attributed to the underlying causes of lactic acidosis. Infections that affect the gastrointestinal tract can cause diarrhea and subsequent dehydration. Dehydration can cause the development of lactic acidosis, implicating diarrhea as a symptom of the condition. Diarrhea is also a common result of bacterial overgrowth into an affected individual's small intestine. Lactic acidosis can be caused by a patient's inability to digest glucose fully from the food that they consume. This allows undigested glucose to proceed into the large intestine, where the good gut bacteria consume it and begin to grow out of control. Glucose consumption by the gut bacteria causes an accumulation of lactic acid because it is a byproduct of the process. This bacteria becomes unmanageable and grows into the small intestine where it causes inflammation and damage to the intestinal lining. The bacteria can also metabolize bile salts into compounds that cannot be reabsorbed by the intestines. This absorption impairment causes the patient to experience diarrhea.

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